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Some aspects of the bonds interlinking soluble fibrin complexes in human plasma.

Thrombosis research

Ly B, Jakobsen E.
PMID: 1114480
Thromb Res. 1975 Jan;6(1):65-73. doi: 10.1016/0049-3848(75)90151-6.

No abstract available.

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Ly B, Jakobsen E. Some aspects of the bonds interlinking soluble fibrin complexes in human plasma. Thromb Res. 1975;6(1):65-73doi: 10.1016/0049-3848(75)90151-6.
Ly, B., & Jakobsen, E. (1975). Some aspects of the bonds interlinking soluble fibrin complexes in human plasma. Thrombosis research, 6(1), 65-73. https://doi.org/10.1016/0049-3848(75)90151-6
Ly, B, and Jakobsen, E. "Some aspects of the bonds interlinking soluble fibrin complexes in human plasma." Thrombosis research vol. 6,1 (1975): 65-73. doi: https://doi.org/10.1016/0049-3848(75)90151-6
Ly B, Jakobsen E. Some aspects of the bonds interlinking soluble fibrin complexes in human plasma. Thromb Res. 1975 Jan;6(1):65-73. doi: 10.1016/0049-3848(75)90151-6. PMID: 1114480.
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OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?.

Thrombosis research

Faille D, Ajzenberg N, de Chaisemartin L, Granger V, Chollet-Martin S, de Raucourt E, Hammel P, Lévy P, Ruszniewski P, Rebours V.
PMID: 27161678
Thromb Res. 2016 Apr;140:S170-1. doi: 10.1016/S0049-3848(16)30123-2. Epub 2016 Apr 08.

INTRODUCTION: Venous thrombo-embolic events (VTE) occur frequently in patients with pancreatic cancer and contribute to elevated morbidity and mortality. Clinical risk factors for thrombosis such as cancer stage and tumor grade have been clearly identified. Recently, several biomarkers have...

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Faille D, Ajzenberg N, de Chaisemartin L, et al. OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thromb Res. 2016;140:S170-1doi: 10.1016/S0049-3848(16)30123-2.
Faille, D., Ajzenberg, N., de Chaisemartin, L., Granger, V., Chollet-Martin, S., de Raucourt, E., Hammel, P., Lévy, P., Ruszniewski, P., & Rebours, V. (2016). OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thrombosis research, 140S170-1. https://doi.org/10.1016/S0049-3848(16)30123-2
Faille, D, et al. "OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?." Thrombosis research vol. 140 (2016): S170-1. doi: https://doi.org/10.1016/S0049-3848(16)30123-2
Faille D, Ajzenberg N, de Chaisemartin L, Granger V, Chollet-Martin S, de Raucourt E, Hammel P, Lévy P, Ruszniewski P, Rebours V. OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thromb Res. 2016 Apr;140:S170-1. doi: 10.1016/S0049-3848(16)30123-2. Epub 2016 Apr 08. PMID: 27161678.
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OC-09 - Microparticle-mediated transfer of TF hypercoagulability between cancer cells.

Thrombosis research

Shaker H, Rothmeier AS, Kirwan CC, Ruf W.
PMID: 27161681
Thromb Res. 2016 Apr;140:S172. doi: 10.1016/S0049-3848(16)30126-8. Epub 2016 Apr 08.

INTRODUCTION: Tissue Factor (TF) promotes apoptosis-resistance and facilitates haematogenous metastasis. Cancer cells release TF-bearing microparticles (TF-MP) which have pro-coagulant activity. TF-MPs from cancer cells induce increase in TF expression and procoagulant activity of recipient endothelial cells. Drug resistance microparticles...

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Shaker H, Rothmeier AS, Kirwan CC, et al. OC-09 - Microparticle-mediated transfer of TF hypercoagulability between cancer cells. Thromb Res. 2016;140:S172doi: 10.1016/S0049-3848(16)30126-8.
Shaker, H., Rothmeier, A. S., Kirwan, C. C., & Ruf, W. (2016). OC-09 - Microparticle-mediated transfer of TF hypercoagulability between cancer cells. Thrombosis research, 140S172. https://doi.org/10.1016/S0049-3848(16)30126-8
Shaker, H, et al. "OC-09 - Microparticle-mediated transfer of TF hypercoagulability between cancer cells." Thrombosis research vol. 140 (2016): S172. doi: https://doi.org/10.1016/S0049-3848(16)30126-8
Shaker H, Rothmeier AS, Kirwan CC, Ruf W. OC-09 - Microparticle-mediated transfer of TF hypercoagulability between cancer cells. Thromb Res. 2016 Apr;140:S172. doi: 10.1016/S0049-3848(16)30126-8. Epub 2016 Apr 08. PMID: 27161681.
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OC-15 - Risk factors for cancer development after idiopathic venous thromboembolism.

Thrombosis research

Cosmi B, Legnani C, Ghirarduzzi A, De Micheli V, Pengo V, Testa S, Poli D, Antonucci E, Prisco D, Tripodi A, Prandoni P, Palareti G.
PMID: 27161686
Thromb Res. 2016 Apr;140:S174. doi: 10.1016/S0049-3848(16)30132-3. Epub 2016 Apr 08.

INTRODUCTION: Idiopathic venous thromboembolism (VTE) is associated with the risk of cancer but the risk factors for cancer development in such patients are still uncertain.AIM: To assess risk factors for the development of cancer after a standard course of...

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Cosmi B, Legnani C, Ghirarduzzi A, et al. OC-15 - Risk factors for cancer development after idiopathic venous thromboembolism. Thromb Res. 2016;140:S174doi: 10.1016/S0049-3848(16)30132-3.
Cosmi, B., Legnani, C., Ghirarduzzi, A., De Micheli, V., Pengo, V., Testa, S., Poli, D., Antonucci, E., Prisco, D., Tripodi, A., Prandoni, P., Palareti, G. (2016). OC-15 - Risk factors for cancer development after idiopathic venous thromboembolism. Thrombosis research, 140S174. https://doi.org/10.1016/S0049-3848(16)30132-3
Cosmi, B, et al. "OC-15 - Risk factors for cancer development after idiopathic venous thromboembolism." Thrombosis research vol. 140 (2016): S174. doi: https://doi.org/10.1016/S0049-3848(16)30132-3
Cosmi B, Legnani C, Ghirarduzzi A, De Micheli V, Pengo V, Testa S, Poli D, Antonucci E, Prisco D, Tripodi A, Prandoni P, Palareti G. OC-15 - Risk factors for cancer development after idiopathic venous thromboembolism. Thromb Res. 2016 Apr;140:S174. doi: 10.1016/S0049-3848(16)30132-3. Epub 2016 Apr 08. PMID: 27161686.
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PO-10 - Lung cancer with tumour thrombosis: a poor prognosticator.

Thrombosis research

Tan ZK, Casipit P, Pandit NS.
PMID: 27161698
Thromb Res. 2016 Apr;140:S180. doi: 10.1016/S0049-3848(16)30143-8. Epub 2016 Apr 08.

INTRODUCTION: Literature has reported tumour thrombus along the inferior vena cava as a result of underlying renal cell carcinoma (RCC) or along the portal vein from hepatocellular carcinoma (HCC). However, tumor thrombus is a rather uncommon complication of a...

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Tan ZK, Casipit P, Pandit NS. PO-10 - Lung cancer with tumour thrombosis: a poor prognosticator. Thromb Res. 2016;140:S180doi: 10.1016/S0049-3848(16)30143-8.
Tan, Z. K., Casipit, P., & Pandit, N. S. (2016). PO-10 - Lung cancer with tumour thrombosis: a poor prognosticator. Thrombosis research, 140S180. https://doi.org/10.1016/S0049-3848(16)30143-8
Tan, Z K, et al. "PO-10 - Lung cancer with tumour thrombosis: a poor prognosticator." Thrombosis research vol. 140 (2016): S180. doi: https://doi.org/10.1016/S0049-3848(16)30143-8
Tan ZK, Casipit P, Pandit NS. PO-10 - Lung cancer with tumour thrombosis: a poor prognosticator. Thromb Res. 2016 Apr;140:S180. doi: 10.1016/S0049-3848(16)30143-8. Epub 2016 Apr 08. PMID: 27161698.
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PO-14 - Tumour expression of coagulation proteases of the aPC pathway - a role in the pathogenesis of gynaecological cancers?.

Thrombosis research

Martin F, Long JC, O'Toole SA, O'Leary JJ, Abu Saadeh F, Gleeson N, Norris LA.
PMID: 27161702
Thromb Res. 2016 Apr;140:S181. doi: 10.1016/S0049-3848(16)30147-5. Epub 2016 Apr 08.

INTRODUCTION: The close relationship between coagulation, thrombosis and cancer has long been established. Gynaecological cancers, in particular ovarian cancers, carry a high risk of thrombosis but coagulation activation is also thought to play a role in tumorigenesis and metastasis....

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Martin F, Long JC, O'Toole SA, et al. PO-14 - Tumour expression of coagulation proteases of the aPC pathway - a role in the pathogenesis of gynaecological cancers?. Thromb Res. 2016;140:S181doi: 10.1016/S0049-3848(16)30147-5.
Martin, F., Long, J. C., O'Toole, S. A., O'Leary, J. J., Abu Saadeh, F., Gleeson, N., & Norris, L. A. (2016). PO-14 - Tumour expression of coagulation proteases of the aPC pathway - a role in the pathogenesis of gynaecological cancers?. Thrombosis research, 140S181. https://doi.org/10.1016/S0049-3848(16)30147-5
Martin, F, et al. "PO-14 - Tumour expression of coagulation proteases of the aPC pathway - a role in the pathogenesis of gynaecological cancers?." Thrombosis research vol. 140 (2016): S181. doi: https://doi.org/10.1016/S0049-3848(16)30147-5
Martin F, Long JC, O'Toole SA, O'Leary JJ, Abu Saadeh F, Gleeson N, Norris LA. PO-14 - Tumour expression of coagulation proteases of the aPC pathway - a role in the pathogenesis of gynaecological cancers?. Thromb Res. 2016 Apr;140:S181. doi: 10.1016/S0049-3848(16)30147-5. Epub 2016 Apr 08. PMID: 27161702.
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PO-20 - Crosstalk between the lectin pathway and haemostasis in patients with pulmonary cancer.

Thrombosis research

Larsen JB, Christensen TD, Hvas CL, Hvas AM.
PMID: 27161707
Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30153-0. Epub 2016 Apr 08.

INTRODUCTION: Recent research has focused on the complement system in cancer, including the lectin pathway of complement activation. Mannose-binding lectin (MBL), a key activator of the lectin pathway, can bind to tumor cell surfaces in vitro, and lectin pathway...

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Larsen JB, Christensen TD, Hvas CL, et al. PO-20 - Crosstalk between the lectin pathway and haemostasis in patients with pulmonary cancer. Thromb Res. 2016;140:S183doi: 10.1016/S0049-3848(16)30153-0.
Larsen, J. B., Christensen, T. D., Hvas, C. L., & Hvas, A. M. (2016). PO-20 - Crosstalk between the lectin pathway and haemostasis in patients with pulmonary cancer. Thrombosis research, 140S183. https://doi.org/10.1016/S0049-3848(16)30153-0
Larsen, J B, et al. "PO-20 - Crosstalk between the lectin pathway and haemostasis in patients with pulmonary cancer." Thrombosis research vol. 140 (2016): S183. doi: https://doi.org/10.1016/S0049-3848(16)30153-0
Larsen JB, Christensen TD, Hvas CL, Hvas AM. PO-20 - Crosstalk between the lectin pathway and haemostasis in patients with pulmonary cancer. Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30153-0. Epub 2016 Apr 08. PMID: 27161707.
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PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status.

Thrombosis research

Vignoli A, Tessarolo S, Marchetti M, Gamba S, Piras F, Finazzi G, van der Meijden PE, Swieringa F, Ten Cate H, Heemskerk JW, Rambaldi A, Falanga A.
PMID: 27161708
Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30152-9. Epub 2016 Apr 08.

INTRODUCTION: The myeloproliferative neoplasms ET and PV are characterized by a high incidence of both arterial and venous thrombosis, and/or microcirculatory disturbances. Three somatic mutations, i.e. JAK2-V617F, Calreticulin (CalR) and MPL, commonly found in these diseases, correlate with different...

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Vignoli A, Tessarolo S, Marchetti M, et al. PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thromb Res. 2016;140:S183doi: 10.1016/S0049-3848(16)30152-9.
Vignoli, A., Tessarolo, S., Marchetti, M., Gamba, S., Piras, F., Finazzi, G., van der Meijden, P. E., Swieringa, F., Ten Cate, H., Heemskerk, J. W., Rambaldi, A., & Falanga, A. (2016). PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thrombosis research, 140S183. https://doi.org/10.1016/S0049-3848(16)30152-9
Vignoli, A, et al. "PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status." Thrombosis research vol. 140 (2016): S183. doi: https://doi.org/10.1016/S0049-3848(16)30152-9
Vignoli A, Tessarolo S, Marchetti M, Gamba S, Piras F, Finazzi G, van der Meijden PE, Swieringa F, Ten Cate H, Heemskerk JW, Rambaldi A, Falanga A. PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30152-9. Epub 2016 Apr 08. PMID: 27161708.
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PO-23 - Expression of heparanase in cancer as biomarker of malignancies: overexpression in an aggressive, poor survival gastric cancer "gastric signet ring cell carcinoma" compared with that of other gastric cancers.

Thrombosis research

Fourgeaud C, Derieux S, Mirshahi S, Besbes S, Chidiac J, Mahé I, Contant G, Pocard M, Soria J, Mirshahi M.
PMID: 27161709
Thromb Res. 2016 Apr;140:S184-5. doi: 10.1016/S0049-3848(16)30156-6. Epub 2016 Apr 08.

INTRODUCTION: Gastric signet ring cell carcinoma (GSRC) is a distinct entity among of other gastric cancer. With unknown etiopathology, their incidence is increasing and it presents a low sensitivity to chemotherapy.AIM: Here, we studied the expression of the heparanase...

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Fourgeaud C, Derieux S, Mirshahi S, et al. PO-23 - Expression of heparanase in cancer as biomarker of malignancies: overexpression in an aggressive, poor survival gastric cancer "gastric signet ring cell carcinoma" compared with that of other gastric cancers. Thromb Res. 2016;140:S184-5doi: 10.1016/S0049-3848(16)30156-6.
Fourgeaud, C., Derieux, S., Mirshahi, S., Besbes, S., Chidiac, J., Mahé, I., Contant, G., Pocard, M., Soria, J., & Mirshahi, M. (2016). PO-23 - Expression of heparanase in cancer as biomarker of malignancies: overexpression in an aggressive, poor survival gastric cancer "gastric signet ring cell carcinoma" compared with that of other gastric cancers. Thrombosis research, 140S184-5. https://doi.org/10.1016/S0049-3848(16)30156-6
Fourgeaud, C, et al. "PO-23 - Expression of heparanase in cancer as biomarker of malignancies: overexpression in an aggressive, poor survival gastric cancer "gastric signet ring cell carcinoma" compared with that of other gastric cancers." Thrombosis research vol. 140 (2016): S184-5. doi: https://doi.org/10.1016/S0049-3848(16)30156-6
Fourgeaud C, Derieux S, Mirshahi S, Besbes S, Chidiac J, Mahé I, Contant G, Pocard M, Soria J, Mirshahi M. PO-23 - Expression of heparanase in cancer as biomarker of malignancies: overexpression in an aggressive, poor survival gastric cancer "gastric signet ring cell carcinoma" compared with that of other gastric cancers. Thromb Res. 2016 Apr;140:S184-5. doi: 10.1016/S0049-3848(16)30156-6. Epub 2016 Apr 08. PMID: 27161709.
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PO-27 - Thrombin generation in pancreatic cancer and multiple myeloma with use of calibrated automated thrombography.

Thrombosis research

Adesanya MA, Maraveyas A, Madden LA.
PMID: 27161715
Thromb Res. 2016 Apr;140:S186. doi: 10.1016/S0049-3848(16)30160-8. Epub 2016 Apr 08.

INTRODUCTION: The calibrated automated thrombography (CAT) assay is emerging as a reliable tool for real time estimation of thrombin generation (TG) potential. As a time-dependent colorimetric assessment of thrombin quantity generated per sample, it measures the amount of thrombin-cleaved...

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Adesanya MA, Maraveyas A, Madden LA. PO-27 - Thrombin generation in pancreatic cancer and multiple myeloma with use of calibrated automated thrombography. Thromb Res. 2016;140:S186doi: 10.1016/S0049-3848(16)30160-8.
Adesanya, M. A., Maraveyas, A., & Madden, L. A. (2016). PO-27 - Thrombin generation in pancreatic cancer and multiple myeloma with use of calibrated automated thrombography. Thrombosis research, 140S186. https://doi.org/10.1016/S0049-3848(16)30160-8
Adesanya, M A, et al. "PO-27 - Thrombin generation in pancreatic cancer and multiple myeloma with use of calibrated automated thrombography." Thrombosis research vol. 140 (2016): S186. doi: https://doi.org/10.1016/S0049-3848(16)30160-8
Adesanya MA, Maraveyas A, Madden LA. PO-27 - Thrombin generation in pancreatic cancer and multiple myeloma with use of calibrated automated thrombography. Thromb Res. 2016 Apr;140:S186. doi: 10.1016/S0049-3848(16)30160-8. Epub 2016 Apr 08. PMID: 27161715.
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PO-28 - Protein C levels are associated with mortality in patients with advanced cancer.

Thrombosis research

Wilts IT, Hutten BA, Meijers JC, Spek CA, Büller HR, Kamphuisen PW.
PMID: 27161716
Thromb Res. 2016 Apr;140:S186-7. doi: 10.1016/S0049-3848(16)30161-X. Epub 2016 Apr 08.

INTRODUCTION: In cancer, tumor progression and metastasis are promoted by prohemostatic activity. Protein C (PC) is involved in hemostasis, inflammation and signal transduction, and has a protective effect on the endothelial barrier. In mice, administration of activated PC reduced...

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Wilts IT, Hutten BA, Meijers JC, et al. PO-28 - Protein C levels are associated with mortality in patients with advanced cancer. Thromb Res. 2016;140:S186-7doi: 10.1016/S0049-3848(16)30161-X.
Wilts, I. T., Hutten, B. A., Meijers, J. C., Spek, C. A., Büller, H. R., & Kamphuisen, P. W. (2016). PO-28 - Protein C levels are associated with mortality in patients with advanced cancer. Thrombosis research, 140S186-7. https://doi.org/10.1016/S0049-3848(16)30161-X
Wilts, I T, et al. "PO-28 - Protein C levels are associated with mortality in patients with advanced cancer." Thrombosis research vol. 140 (2016): S186-7. doi: https://doi.org/10.1016/S0049-3848(16)30161-X
Wilts IT, Hutten BA, Meijers JC, Spek CA, Büller HR, Kamphuisen PW. PO-28 - Protein C levels are associated with mortality in patients with advanced cancer. Thromb Res. 2016 Apr;140:S186-7. doi: 10.1016/S0049-3848(16)30161-X. Epub 2016 Apr 08. PMID: 27161716.
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OC-16 - Neutrophil extracellular traps and tissue factor-bearing microvesicles: a liaison dangereuse causing overt DIC in cancer patients?.

Thrombosis research

Hell L, Thaler J, Martinod K, Ay C, Posch F, Wagner DD, Pabinger I.
PMID: 27161688
Thromb Res. 2016 Apr;140:S174-5. doi: 10.1016/S0049-3848(16)30133-5. Epub 2016 Apr 08.

INTRODUCTION: Overt disseminated intravascular coagulation (DIC) is a systemic process characterized by excessive coagulation activation and fibrinolysis that may occur in cancer patients. The underlying pathomechanisms are still poorly understood. Recent experimental studies found an important role for the...

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Hell L, Thaler J, Martinod K, et al. OC-16 - Neutrophil extracellular traps and tissue factor-bearing microvesicles: a liaison dangereuse causing overt DIC in cancer patients?. Thromb Res. 2016;140:S174-5doi: 10.1016/S0049-3848(16)30133-5.
Hell, L., Thaler, J., Martinod, K., Ay, C., Posch, F., Wagner, D. D., & Pabinger, I. (2016). OC-16 - Neutrophil extracellular traps and tissue factor-bearing microvesicles: a liaison dangereuse causing overt DIC in cancer patients?. Thrombosis research, 140S174-5. https://doi.org/10.1016/S0049-3848(16)30133-5
Hell, L, et al. "OC-16 - Neutrophil extracellular traps and tissue factor-bearing microvesicles: a liaison dangereuse causing overt DIC in cancer patients?." Thrombosis research vol. 140 (2016): S174-5. doi: https://doi.org/10.1016/S0049-3848(16)30133-5
Hell L, Thaler J, Martinod K, Ay C, Posch F, Wagner DD, Pabinger I. OC-16 - Neutrophil extracellular traps and tissue factor-bearing microvesicles: a liaison dangereuse causing overt DIC in cancer patients?. Thromb Res. 2016 Apr;140:S174-5. doi: 10.1016/S0049-3848(16)30133-5. Epub 2016 Apr 08. PMID: 27161688.
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